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Abstract ’l’oxoplaamosia 106 the term app.lied to aoquired and. congen1 tal iniac tiona 0;[ DIan and. 0thl:U’ ij!;!lmmal e caused. tly the p.robozoonpuae1 ue ’J.’oxoplnswa go.ng,U.+ It ls e.widely dietF1buted ~ono ”ie 1nfeo tion wi th d,iffareot manifests tions (Wrigbt, Tbere was an 1nt’.eI’V1Uof about ,30 years between the detaction of ’fuxoplll.ama gond1.1 and tbe deacr1ptlo11 of? tue tlra1i clinically diagnosed. eaae of oongeni tal toxoPl8SQIOsis in man. ToxoolfU!!ll.8Was first del;ectied by NlcoJ,le and Maneeaur 10 l.YO~ in a north African rodent, Cte:ooda~I;Ylus gond~i sod in the sallie year ;i Do arab bi t by S,pl eMot’E! II 908) in ar 8.1’.11. 1n re trospect, toe first .daac~iption of th~ disease in man was by Jaoku. an o,Pl:Itllallllologiatr in. 1923. He deaor1bedl tbe organism but waa u.nabla to identUy 1t. ”l’he 1’i.rst iaolation of Tompla~ SQndi1 :trom 8. huwan pe:t.:l.8nt (JleYi’born infant) was l’epo~ted by Wo~f ;!.!~. (.1939). SUbsequent illlllJUnO.l(lg1oal and b;iologioal studies eatabl1shed ttle 1dent1ty of the cau.aat1 ve agent. 0 thar early J’”ports Q·f human 1nfeo tiona include those- (If P:l.Xl.ltertCln and We1nman llY40) I Sabin (194-1) and P1Dk:erton $lld. Henciel”son l19~1). |