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العنوان
statistical anaiysis of ethanol toxicity in egybt(1980-1985/)
الناشر
mohamed ahmed el-shishtawy,
المؤلف
el-shishtawy,mohamed ahmed
هيئة الاعداد
باحث / mohamed ahmed el-shishtawy
مشرف / alia ahmed el-nagdy
مشرف / mohmed kamel ahmed
مشرف / Waffia Zein Al-Abdin Atta
مناقش / alia ahmed el-nagdy
مناقش / mohmed kamel ahmed
الموضوع
fornesic toxicoloo
تاريخ النشر
1986 .
عدد الصفحات
91p;.
اللغة
الإنجليزية
الدرجة
ماجستير
التخصص
علم الأمراض والطب الشرعي
تاريخ الإجازة
1/1/1986
مكان الإجازة
جامعة بنها - كلية طب بشري - الطب الشرعي والسموم
الفهرس
Only 14 pages are availabe for public view

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Abstract

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SUMMARY AND CONCLUSION
Ethyl alcohol is a volatile and flammable liquid
which enters in many beverages. Its concntration in
these beverages differes from one drink to
It is least in beer 2-5%.
It is easly absorbed from the mucous membrane. Hince
another.
about 20% are absorbed from the stomach and the intestinal
tract absorbs the remainder.
Ethanol is oxidized in the liver into acetaldehyde
by enzyme alcohol dehydrogenase (ADH), then acetic acid
and lastly CO2 and water. Hence metabolic acidosis may
occur.
Ethanol intoxication may be harmful to almost any
system. However, it affects mainly the C.N.S and the
liver.
The central nervous system effects include acute behavioural
changes involving acute alcoholic intoXication,
alcoholic coma, withdrawal syndromes, nutritional disorders
and consequences of alcoholic liver diseases.
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Acute behavioral changes involve acute intoriction,
alcoholic coma, hang over and amnestic episodes.
Acute Alcoholic intoxication may occur with blood
alcohol level as low as IOOmg/IOOmI and death may occur
at blood alcohol level 40Omg/IOOmI. It may reach high
up to TOOmg!IOOmI. in alcoholics.
There is a wide spectrum of manifestations of alcohoI
with drawal ranging from anxicty and tremulocuness
to full blown delerium tremenes. The later is an acute
organic psychosis that is usually manifested within
24-72 hours after the last drink.Chronic use of alcohol
produces nutritional defeciencies resu1ting in certain
characteristics C.N.S manifestations. The most common
of these are alcoholic dementia, Wernicke’s encephalopathy,
Korsakoff’s psychosis and alcoholic polyneuropathy.
Wernicke’s encephalopathy is manifested by ataxia
ophthalmoplegia and mental confusion, It is an acute disorder
due to thiamine deffeciency. It may be followed
by Korsakoff’s psychosis where the cloudness of consciousness
anterograde and retograde amnesia.
The effects of alcohol on the liver result not only